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Growth factors such as (e.g., VEGF, platelet-derived growth factor [PDGF] bind to a receptor tyrosine kinase (RTK) on the cell membrane, causing autophosphorylation of the receptor, thus creating docking sites for intracellular signaling proteins

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Treatment of human induced pluripotent stem (iPS) cell-derived macrophage precursor cells (MPCs) 35 with erucamide led to significantly higher levels of PDGF, VEGF, FGF-2, IGF-1 and BDNF compared to controls (Fig