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Excessive release of glutamate and its binding with NMDA, -amino-3-hydroxy-5-methyl-4-isoxazolepropionic acid (AMPA), or kainite (KA) receptors promotes excitotoxicity cell injury with a consequent activation of many molecular pathways, resulting in intracellular calcium (Ca 2+ ) overload 90,91 , reactive oxygen species (ROS) production, mPT, and cell swelling with brain edema 92

We call for more comprehensive studies to deepen our understanding of how PCD influences ovarian reserve function in both physiological and pathological conditions

Hyperglycemic kidney injury triggers the release of proinflammatory and inflammatory cytokines from various cellular types including endothelial cells, podocytes, mesangial cells, and tubular epithelial cells [26]

Background Section 5506 of the Affordable Care Act (Pub

[3] Hashemzaei, M., Fanoudi, S., Rezaei, H., Musavi, S