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7 Therapeutic strategies and clinical translation With the deepening understanding of ferroptosis mechanisms, an increasing number of molecular targets have been identified as closely associated with the pathological progression of AD

Excess copper in cells activates transcription factors, upregulates ATG5 expression, and modulates the AMPK-mTOR pathway, thereby inducing autophagy (Xue et al., 2023a

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All other animals were transcardially perfused with Tyrodes solution followed by 4% paraformaldehyde in 0.1 M phosphate buffer (pH 7.4)