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Sancar, A., Lindsey-Boltz, L

In rabbit studies with 0.8 cm radial bone defects that remained incompletely healed in all control animals for 6 weeks, BPC-157 treatment (administered locally into the bone defect or intramuscularly) produced healing comparable to standard treatments including local bone marrow application or autologous cortical bone grafting

NNMT inhibition increases cellular concentrations of both substrates that the enzyme would otherwise consume: SAM (the universal methyl donor) and nicotinamide (the NAD + salvage precursor)

Head-to-Head Comparison Tendon and Ligament Repair Both peptides accelerate tendon healing, but through different pathways

It also shows potential to reduce inflammatory markers like IL-6 and CRP, helping researchers understand obesity-related inflammation