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All these effects can partly be explained through the impact of OS, caused by the inequality between ROS production and antioxidant defense mechanisms (Figure 5) (19, 153)

These findings suggest that in experimental uveitis, the severity of ocular inflammation and tissue damage may be mediated by hydroxyl radicals catalyzed by iron, thus deferoxamine mesylate can serve as an anti-inflammatory agent

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In Section Effects of polymorphisms in glucoronosyl transferases we showed that polymorphisms in the glucoronosyl transferase enzymes can have a large effect on the amount of liver damage caused by moderate overdoses of APAP

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