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Under metabolic stress, MOTS-c translocates from the cytoplasm to the nucleus, directly regulating gene expression related to stress adaptation

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Oral bioavailability has not been characterized, and the compound's heptapeptide structure (773.89 Da) makes oral activity less likely than for the smaller Dihexa (504.66 Da), though not impossible given its modified sequence

Treatment reduces ferroptosis levels in these cells and improves the condition, highlighting the importance of anti-ferroptosis strategies in the disease