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In fulfilling this protective role, reduced glutathione is converted into oxidized glutathione, at which point it becomes inactive

Abnormalities of RCD pathways in mature erythrocytes are well-documented for inherited blood disorders

These typically resolve as the body adapts

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The state of hepatic GH resistance in anorexia nervosa is further corroborated by the lack of increase in IGF-I following administration of supraphysiological doses of exogenous GH in women with anorexia nervosa (61), and only refeeding appears to able to restore IGF-I levels (56, 59, 62)