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In this regard, Zydan et al., in an animal model of the relapsing-remitting experimental autoimmune encephalomyelitis, showed that ALC as a supplementary treatment to dexamethasone results in a significant reduction in MDA and caspase-3 activity and a significant elevation in Bcl-2 and GSH expression and consequently provides considerable antiapoptotic and antioxidant milieu in the spinal cord and brain milieu [47]

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[PubMed ID: 17565645].

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This structure enhances MT-II affinity to the melanocortin receptor 3 (MC3R) and melanocortin receptor 4 (MC4R), in addition to MC1R [6]