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In the CAE model, pharmacological inhibition of citrullination is a potent suppressor of inflammatory demyelination after an immune stimulation, in the absence of any direct anti-inflammatory effect by the inhibitors themselves, indicating that proteins modified in this manner can be potent antigenic targets (Caprariello et al., 2018)

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Tom Brunner: As youve mentioned, glaucoma is a very complex disease, which I think is why were still searching for ways to cure it or to, as you say, to reset the cells, and that does raise another question in terms of other some of the different sub types of glaucoma

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