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Finally, GSH availability negatively regulates SLC25A39 protein abundance, coupling redox homeostasis to mitochondrial GSH import in mammalian cells
Dequan Xus lab, Huazhong Agricultural University) and pEGFP-C1 and pmCherry-C1 vectors (gifted from Prof
The cold-stress regulation of BAM3 is mainly linked to S -glutathionylation of the Cys 433 residue, while two other Cys residues found near the catalytic site also undergo S -glutathionylation and could participate in inhibition of activity as well [267]
Nat Rev Cancer 16:447462 Eriksson SE, Ceder S, Bykov VJN, Wiman KG (2019) p53 as a hub in cellular redox regulation and therapeutic target in cancer
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