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Acetaminophen toxicity results in site-specific mitochondrial damage in isolated mouse hepatocytes

Food and Agriculture Organization of the United Nations

On the other hand, PGC-1/NRF1 regulates FUNDC1-mediated mitophagy through transcription, promoting mitochondrial biogenesis during the process of mitochondrial autophagy, accelerating mitochondrial turnover, and maintaining normal mitochondrial respiration and mitochondrial population stability

Furthermore, ZLN005 treatment activates mitophagy, as manifested by increased expression of LC3-II and co-localization between LC3 and mitochondria, and the protective effects are abrogated in TFEB-knockdown cells, suggesting that PGC-1 activation improves mitochondrial dysfunction via TFEB-mediated autophagy

Improves Sleep Quality: Enjoy better rest with reduced oxidative stress