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Well-defined and clinically relevant genetic polymorphisms in both phase I and phase II drug-metabolizing enzymes exist that result in altered efficacy of drug therapy or adverse drug reactions (ADRs)

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Not only did both groups show significant improvements in bilirubin levels (a substance made in the liver during the normal process of breaking down red blood cells that a healthy liver regularly eliminates) months after the trial ended, they also saw a reduction of malondialdehyde, a marker of cell damage in the liver