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Copper binding is what activates most of the documented biological effects strong Cu(II) chelators such as bathocuproine, which strip the copper from GHK-Cu, abolish its actions on collagen synthesis, wound healing, and gene expression in published in-vitro assays [3]

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Because GHK-Cu's anti-inflammatory action is selective rather than broadly suppressive, it calms rosacea-prone and reactive skin without the barrier-thinning tradeoffs of steroid-based approaches