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doi: 10.1016/j.neubiorev.2015.07.015 419 MissailidisDSanislavOAllanCYAnnesleySJFisherPR

A compound that produces identical protective effects whether administered systemically or topically to the target tissue suggests both a circulating effector mechanism and a local tissue-level mechanism, consistent with the dual NO-VEGF angiogenic pathway described above

Mechanism of action BPC-157 activates the FAK-paxillin pathway in cells

GHK-Cu is extensively studied for its ability to stimulate collagen production, reduce oxidative stress, and enhance wound healing

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