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66 It also inhibits the activation of nuclear factor erythroid 2-related factor 2 (NRF2), exacerbating oxidative stress injury to the kidneys

This highlights the potential to exploit HO-1 as a valuable target for therapeutic intervention in DFU cases (Fig

Researchers planning injection studies should recognise that mechanism and route are decoupled in the current evidence base

To test this hypothesis, mice were sacrificed at 72 hours after APAP administration (300 mg/kg) and livers were harvested for BrdU, Ki67 and PCNA staining

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