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The dominant inflammatory phenotype in allergic (atopic) asthma is Type 2 (T2)-high , driven by Th2 lymphocytes, ILC2 cells, and key cytokines IL-4, IL-5, IL-13, and TSLP

We had previously reported that SAECs are more susceptible to cigarette smoke extract than large airway epithelial cells [29]

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The consultation is open, and public health researchers, clinicians, cessation services, and patient organisations across Europe all have a role to play