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Sequeira DB, Diogo P, Gomes BPFA, Pea J, Santos JMM

Any immune or inflammatory settings in which the amount or activity of any of these proteins is lowered or does not increase appropriately (as we modeled with erastin) could dramatically perturb homeostatic balance and enhance T2 inflammatory factor expression and oxidatively driven cell death

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In this regard, inappropriate activation of estrogen receptor alpha signaling concomitantly with the elevation of testosterone has been demonstrated to trigger prostate cancer and prostate hyperplasia in mice [27]