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Genetically susceptible follicles, particularly those at the crown and temples, possess higher concentrations of androgen receptors, making them more vulnerable to DHT-induced damage

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A less common, but more severe, form of complete BPI is where all parts of the brachial plexus are pulled out of the spinal cord

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NIX/BNIP3L has been shown to be involved in the PINK1-parkin pathway, being ubiquitinated by parkin and recruiting the mitochondrial phagocytosis receptor, NBR1, for mitochondrial degradation [17]