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Gupta AK, Venkataraman M, Talukder M, Bamimore MA

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In the present study, exogenous GSH enhanced JA signal transduction by inhibiting the expression of genes related to the JAZ protein, thus increasing the levels of JA

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Mouse T-lymphoma cells transfected with the human ABCB1 gene, which overexpress P-gp, when treated with the substituted naringenin derivatives together with doxorubicin, reversed drug resistance substantially